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By James M. Gruschus, Author
Unlike other neurodegenerative diseases such as Alzheimer’s in chimpanzees, multiple sclerosis in macaques, or Lou Gehrig’s disease in dogs, Parkinson’s disease has no known animal parallel. This intriguing fact has led researchers to delve deeper into the causes of Parkinson’s disease and how the evolution of the human brain has made us uniquely vulnerable.
Genetic risk factors and environmental toxins play a significant role in the development of Parkinson’s disease. The interplay between these factors and neuroanatomy is a complex one, involving subatomic physics, socioeconomics, and more. This multidisciplinary approach aims to understand the intricate connections between neurodegenerative disease, neural pathways, and innate immunity.
Synuclein and the Coelacanth: The Molecular and Evolutionary Origins of Parkinson’s Disease presents a new hypothesis on the origins of Parkinson’s disease. This hypothesis proposes that the unique vulnerability of humans to Parkinson’s disease is a result of our brain’s evolution, making us distinct from other species.
Currently, researchers are working on developing new therapeutic agents that aim to go beyond just treating the symptoms of Parkinson’s disease. These new treatments hope to halt the progression of the disease, offering new hope for patients affected by this debilitating condition.
For more information on Parkinson’s disease, its causes, and the latest research, visit our dedicated health and science section.
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